Growth arrest-specific gene 2 suppresses hepatocarcinogenesis by intervention of cell cycle and p53-dependent apoptosis

Figure 2 Effect of knockdown on endogenous GAS2 in MIHA cells. A: Western blot analysis of siGAS2, β-actin was used as the loading control; B: Cell counting (^aP < 0.05 vs control); C: Cell viability (^aP < 0.05 vs control); D: Anchorage-dependent colony formation (^bP < 0.01 vs control). GAS2: Growth arrest-specific gene 2.