Subclinical atherosclerosis is linked to small intestinal bacterial overgrowth via vitamin K2-dependent mechanisms

doi:10.3748/wjg.v23.i7.1241

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Feb 21, 2017; 23(7): 1241-1249
Published online Feb 21, 2017. doi: 10.3748/wjg.v23.i7.1241

Table 2 Comparison of circulating levels of dephosphorylated-uncarboxylated matrix Gla-protein and ultrasound parameters (pulse-wave velocity, intima-media thickness, and vascular calcifications) in patients with or without small intestinal bacterial overgrowth

	Overall	SIBO (n = 12)	no-SIBO (n = 27)	P value
dp-ucMGP (μg/L)	4.73 (1.75-23.8)	9.5 (3.6-23.8)	4.2 (1.75-14.5)	0.004^a
PWV (m/s)	8.5 (5.7-14)	10.25 (5.7-14)	7.68 (5.6-11)	0.002^a
IMT (μm)	661 (467-1009)	596 (467-1009)	663 (506-932)	0.465
Calcifications	16 (41)	6 (50)	10 (37)	0.609

Data are expressed as median (range) or frequency (%), significant comparisons (^aP < 0.05). SIBO: Small intestinal bacterial overgrowth; dp-ucMGP: Dephosphorylated-uncarboxylated matrix Gla-protein; PWV: Pulse-wave velocity; IMT: Intima-media thickness.

Citation: Ponziani FR, Pompili M, Di Stasio E, Zocco MA, Gasbarrini A, Flore R. Subclinical atherosclerosis is linked to small intestinal bacterial overgrowth via vitamin K2-dependent mechanisms. World J Gastroenterol 2017; 23(7): 1241-1249
URL: https://www.wjgnet.com/1007-9327/full/v23/i7/1241.htm
DOI: https://dx.doi.org/10.3748/wjg.v23.i7.1241