Remote ischemic perconditioning prevents liver transplantation-induced ischemia/reperfusion injury in rats: Role of ROS/RNS and eNOS

doi:10.3748/wjg.v23.i5.830

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Feb 7, 2017; 23(5): 830-841
Published online Feb 7, 2017. doi: 10.3748/wjg.v23.i5.830

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Figure 6 Effects of remote ischemic perconditioning on endothelial nitric oxide synthase and nitric oxide expression. A: Western blots of phospho-ser1176-endothelial nitric oxide synthase (eNOS) [p-eNOS] and total eNOS (t-eNOS) detection in liver tissues (gels run under the same experimental conditions); B: Statistical analysis of phospho-ser1176-eNOS and total eNOS in liver tissues; C: mRNA level of eNOS; D: Total nitrate/nitrite content in the four groups. Data represent mean ± SEM for 5 animals per group. ^aP < 0.05 vs sham group; ^bP < 0.05 vs orthotopic liver transplantation (OLT) group.

Citation: He N, Jia JJ, Li JH, Zhou YF, Lin BY, Peng YF, Chen JJ, Chen TC, Tong RL, Jiang L, Xie HY, Zhou L, Zheng SS. Remote ischemic perconditioning prevents liver transplantation-induced ischemia/reperfusion injury in rats: Role of ROS/RNS and eNOS. World J Gastroenterol 2017; 23(5): 830-841
URL: https://www.wjgnet.com/1007-9327/full/v23/i5/830.htm
DOI: https://dx.doi.org/10.3748/wjg.v23.i5.830