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©The Author(s) 2017.
World J Gastroenterol. Sep 21, 2017; 23(35): 6403-6411
Published online Sep 21, 2017. doi: 10.3748/wjg.v23.i35.6403
Published online Sep 21, 2017. doi: 10.3748/wjg.v23.i35.6403
Figure 3 Measures of phospho-mTOR/mTOR levels (A) and phospho-S6K1/S6K1 (B) in gastric mucosa and representative Western blots from animals in the fasting state treated with i.
p. rimonabant or vehicle (n = 9). Measures of phospho-mTOR/mTOR levels (C) and phospho-S6K1/S6K1 (D) in gastric mucosa and representative Western blots from animals in the fasting state treated with i.p. AM281 (n = 6) or vehicle (n = 6). β-actin was used as a loading control. Dividing lines indicate splicings within the same gel. The results are expressed as percentages over control, aP < 0.05.
- Citation: Folgueira C, Barja-Fernandez S, Prado L, Al-Massadi O, Castelao C, Pena-Leon V, Gonzalez-Saenz P, Baltar J, Baamonde I, Leis R, Dieguez C, Pagotto U, Casanueva FF, Tovar SA, Nogueiras R, Seoane LM. Pharmacological inhibition of cannabinoid receptor 1 stimulates gastric release of nesfatin-1 via the mTOR pathway. World J Gastroenterol 2017; 23(35): 6403-6411
- URL: https://www.wjgnet.com/1007-9327/full/v23/i35/6403.htm
- DOI: https://dx.doi.org/10.3748/wjg.v23.i35.6403