Hepatitis B virus infection and alcohol consumption

doi:10.3748/wjg.v23.i15.2651

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Apr 21, 2017 (publication date) through Feb 8, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Apr 21, 2017; 23(15): 2651-2659
Published online Apr 21, 2017. doi: 10.3748/wjg.v23.i15.2651

Table 3 Genetic polymorphisms associated with increased risk of alcoholic liver disease progression

Gene	Polymorphism	Reported association	Ref.
ADH	ADH21/2	Decrease the rate of ethanol metabolism	[47]
ADH	ADH21/1	Decrease the rate of ethanol metabolism	[47]
ALDH	ALDH22/2	Increase alcohol sensitivity	[47]
ALDH	ALDH21/2	Increase alcohol sensitivity	[47]
CYP2E1	C1/C2	Increase free radical generation, lipid peroxidation, and fatty change	[9]
CYP2E1	C2/C2		[9]
PNPLA3	rs738409C>G	Increase the risk of liver cirrhosis and HCC	[48,49]
CD14	159TT	Enhance inflammatory responses	[50]
CD14	159TT	Develop alcoholic liver disease	[50]
TNF-α	238G>A	Develop alcoholic liver disease	[51]

ADH: Alcohol dehydrogenase; ALDH: Acetaldehyde dehydrogenase; CYP2E1: Cytochrome P450 2E1; HCC: Hepatocellular carcinoma; PNPLA3: Patatin-like phospholipase domain containing 3; TNF-α: Tumor necrosis factor-α.

Citation: Iida-Ueno A, Enomoto M, Tamori A, Kawada N. Hepatitis B virus infection and alcohol consumption. World J Gastroenterol 2017; 23(15): 2651-2659
URL: https://www.wjgnet.com/1007-9327/full/v23/i15/2651.htm
DOI: https://dx.doi.org/10.3748/wjg.v23.i15.2651