HER2-induced metastasis is mediated by AKT/JNK/EMT signaling pathway in gastric cancer

doi:10.3748/wjg.v22.i41.9141

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Nov 7, 2016; 22(41): 9141-9153
Published online Nov 7, 2016. doi: 10.3748/wjg.v22.i41.9141

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Figure 7 Effect of JNK or AKT on cell growth of human epidermal growth factor receptor 2-positive gastric cancer cell lines. A, C: GC cell lines were treated with either DMSO or SP600125 (SP); B, D: Cells were treated with either DMSO or LY294002 (LY). Cell growth rates were analyzed using crystal violet assay on the indicated times, and absorbance was measured. Results were calculated as percentages relative to DMSO vehicle control. Data are expressed as mean ± SD (n = 4 per each group). ^aP < 0.05 vs DMSO vehicle control.

Citation: Choi Y, Ko YS, Park J, Choi Y, Kim Y, Pyo JS, Jang BG, Hwang DH, Kim WH, Lee BL. HER2-induced metastasis is mediated by AKT/JNK/EMT signaling pathway in gastric cancer. World J Gastroenterol 2016; 22(41): 9141-9153
URL: https://www.wjgnet.com/1007-9327/full/v22/i41/9141.htm
DOI: https://dx.doi.org/10.3748/wjg.v22.i41.9141