HER2-induced metastasis is mediated by AKT/JNK/EMT signaling pathway in gastric cancer

doi:10.3748/wjg.v22.i41.9141

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Nov 7, 2016; 22(41): 9141-9153
Published online Nov 7, 2016. doi: 10.3748/wjg.v22.i41.9141

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Figure 6 Synergistic effect of JNK and AKT on metastatic potential of HER2-positive gastric cancer cells. Combined effects of AKT and JNK on the metastatic potential of SNU-216 and NCI-N87 were determined by Transwell migration assay. A, B: Cells were treated with DMSO or SP600125 (SP) in the presence or absence of LY294002 (LY). Results were calculated as percentages relative to DMSO vehicle control. Data are expressed as mean ± SD (n = 4 per each group). ^aP < 0.05 vs DMSO vehicle control.

Citation: Choi Y, Ko YS, Park J, Choi Y, Kim Y, Pyo JS, Jang BG, Hwang DH, Kim WH, Lee BL. HER2-induced metastasis is mediated by AKT/JNK/EMT signaling pathway in gastric cancer. World J Gastroenterol 2016; 22(41): 9141-9153
URL: https://www.wjgnet.com/1007-9327/full/v22/i41/9141.htm
DOI: https://dx.doi.org/10.3748/wjg.v22.i41.9141