Endothelial dysfunction in inflammatory bowel diseases: Pathogenesis, assessment and implications

doi:10.3748/wjg.v22.i3.1067

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jan 21, 2016; 22(3): 1067-1077
Published online Jan 21, 2016. doi: 10.3748/wjg.v22.i3.1067

Table 2 Methods of endothelium dysfunction assessment

Proteins activated by normal and activated endothelium	Cell-based methods
Angiopoietin-1	Circulating angiogenic cells
Angiopoietin-2	Endothelial progenitor cells
Asymmetric dimethylarginine (ADMA)	Endothelial microparticles
Disintegrin and metalloproteinase with thrombospondin motif-13 (ADAMTS13)
Intercellular adhesion molecule-1 (CAM-1)
Plasminogen-activator inhibitor (PAI-1)
Selectins P and E
Tissue plasminogen activator (t-PA)
Thrombomodulin
Vascular adhesion molecule-1 (VCAM-1)
Vascular endothelial growth factor (VEGF)
von Willebrand factor (vWF)
Non-invasive physical methods
Carotis intima-media thickness (cIMT)
Flow mediated dilatation (FMD)
Pulse arterial tonometry (PAT)
Pulse wave analysis (PWA)
Pulse wave velocity (PWV)
Reactive hyperaemia index (RHI)

Citation: Cibor D, Domagala-Rodacka R, Rodacki T, Jurczyszyn A, Mach T, Owczarek D. Endothelial dysfunction in inflammatory bowel diseases: Pathogenesis, assessment and implications. World J Gastroenterol 2016; 22(3): 1067-1077
URL: https://www.wjgnet.com/1007-9327/full/v22/i3/1067.htm
DOI: https://dx.doi.org/10.3748/wjg.v22.i3.1067