Copyright
©The Author(s) 2016.
World J Gastroenterol. May 28, 2016; 22(20): 4881-4890
Published online May 28, 2016. doi: 10.3748/wjg.v22.i20.4881
Published online May 28, 2016. doi: 10.3748/wjg.v22.i20.4881
Figure 7 miR-29a targets TNFRSF1A.
A: The predicted miR-29a binding sites within the 3’UTR of TNFRSF1A and mutant version generated by site mutagenesis are shown; B: Luciferase activity was determined 48 h after transfection. The ratio of normalized sensor to control luciferase activity is shown. Data are shown as the mean ± SD and were obtained from three independent experiments performed in triplicate (aP < 0.05 vs control miR-transfected cells).
- Citation: Fu Q, Qin T, Chen L, Liu CJ, Zhang X, Wang YZ, Hu MX, Chu HY, Zhang HW. miR-29a up-regulation in AR42J cells contributes to apoptosis via targeting TNFRSF1A gene. World J Gastroenterol 2016; 22(20): 4881-4890
- URL: https://www.wjgnet.com/1007-9327/full/v22/i20/4881.htm
- DOI: https://dx.doi.org/10.3748/wjg.v22.i20.4881