Contribution of mammalian target of rapamycin in the pathophysiology of cirrhotic cardiomyopathy

doi:10.3748/wjg.v22.i19.4685

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501

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308

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3715

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290

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May 21, 2016 (publication date) through Dec 30, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. May 21, 2016; 22(19): 4685-4694
Published online May 21, 2016. doi: 10.3748/wjg.v22.i19.4685

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Figure 4 Left ventricular tumor necrosis factor-α levels in control and CCl₄-induced cirrhotic rats treated with normal saline or rapamycin (2 mg/kg). The data are expressed as the mean ± SD. ^eP < 0.001 vs control/normal saline group; ^fP < 0.001 vs cirrhosis/normal saline group. TNF-α: Tumor necrosis factor-α.

Citation: Saeedi Saravi SS, Ghazi-Khansari M, Ejtemaei Mehr S, Nobakht M, Mousavi SE, Dehpour AR. Contribution of mammalian target of rapamycin in the pathophysiology of cirrhotic cardiomyopathy. World J Gastroenterol 2016; 22(19): 4685-4694
URL: https://www.wjgnet.com/1007-9327/full/v22/i19/4685.htm
DOI: https://dx.doi.org/10.3748/wjg.v22.i19.4685