CacyBP/SIP nuclear translocation regulates p27Kip1 stability in gastric cancer cells

doi:10.3748/wjg.v22.i15.3992

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Apr 21, 2016; 22(15): 3992-4001
Published online Apr 21, 2016. doi: 10.3748/wjg.v22.i15.3992

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Figure 8 Interaction between Skp1 and calcyclin binding protein/Siah-1 (1-72) in SGC7901-calcyclin binding protein/Siah-1si transfected with pEGFP/C1-calcyclin binding protein/Siah-1 (1-72). Nuclear protein extracts from cells transfected with pEGFP/C1-CacyBP/SIP or pEGFP/C1- CacyBP/SIP (1-72) after treatment with 10-8 mol/L gastrin were immunoprecipitated with anti-GFP antibody or control IgG. Immune complexes were analyzed by immunoblotting using an anti-Skp1/GFP antibody with ECL-based detection. CacyBP/SIP: Calcyclin binding protein/Siah-1 interacting protein.

Citation: Niu YL, Li YJ, Wang JB, Lu YY, Liu ZX, Feng SS, Hu JG, Zhai HH. CacyBP/SIP nuclear translocation regulates p27Kip1 stability in gastric cancer cells. World J Gastroenterol 2016; 22(15): 3992-4001
URL: https://www.wjgnet.com/1007-9327/full/v22/i15/3992.htm
DOI: https://dx.doi.org/10.3748/wjg.v22.i15.3992