Dihydromyricetin alleviates carbon tetrachloride-induced acute liver injury via JNK-dependent mechanism in mice

doi:10.3748/wjg.v21.i18.5473

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. May 14, 2015; 21(18): 5473-5481
Published online May 14, 2015. doi: 10.3748/wjg.v21.i18.5473

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Figure 5 Dihydromyricetin protects from CCl₄-induced acute liver failure by up-regulating JNK activation both in liver injury mice and acute liver failure mice. A: Western blots depicting JNK pathway proteins on day 2 after CCl₄ (1 mL/kg) injection; B: Western blots depicting JNK pathway proteins on day 2 after CCl₄ (2.6 mL/kg) injection with or without of SP600125 treatment; C: The survival curves of the different conditions (n = 30/group).

Citation: Xie J, Liu J, Chen TM, Lan Q, Zhang QY, Liu B, Dai D, Zhang WD, Hu LP, Zhu RZ. Dihydromyricetin alleviates carbon tetrachloride-induced acute liver injury via JNK-dependent mechanism in mice. World J Gastroenterol 2015; 21(18): 5473-5481
URL: https://www.wjgnet.com/1007-9327/full/v21/i18/5473.htm
DOI: https://dx.doi.org/10.3748/wjg.v21.i18.5473