Dismicrobism in inflammatory bowel disease and colorectal cancer: Changes in response of colocytes

doi:10.3748/wjg.v20.i48.18121

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Dec 28, 2014 (publication date) through Jul 22, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Dec 28, 2014; 20(48): 18121-18130
Published online Dec 28, 2014. doi: 10.3748/wjg.v20.i48.18121

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Figure 1 One hypothesis on the initiation of the inflammatory process in inflammatory bowel disease bases on an abnormal Th1 response mediated by the secretion of interferon gamma and tumor necrosis factor-α while the chronic inflammation is supported by interleukin-5 and interleukin-13 secreted by Th2 cells. IFN: Interferon; TNF: Tumor necrosis factor; IL: Interleukin; IBD: Inflammatory bowel disease.

Citation: Tomasello G, Tralongo P, Damiani P, Sinagra E, Trapani BD, Zeenny MN, Hussein IH, Jurjus A, Leone A. Dismicrobism in inflammatory bowel disease and colorectal cancer: Changes in response of colocytes. World J Gastroenterol 2014; 20(48): 18121-18130
URL: https://www.wjgnet.com/1007-9327/full/v20/i48/18121.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i48.18121