Hepatitis C virus-mediated angiogenesis: Molecular mechanisms and therapeutic strategies

doi:10.3748/wjg.v20.i42.15467

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World Journal of Gastroenterology

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1007-9327

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World J Gastroenterol. Nov 14, 2014; 20(42): 15467-15475
Published online Nov 14, 2014. doi: 10.3748/wjg.v20.i42.15467

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Figure 1 Representative model for tumor angiogenesis induced by hepatocytes growth factor/Scater factor-Met signaling. Intrinsically hepatocytes growth factor/Scater factor (HGF/SF) activates Met receptor on the surface of host endothelial cells leading to cell proliferation and migration. Extrinsically, HGF/SF-Met signaling turns on the angiogenic switch by simultaneous upregulation of pro-angiogenic vascular endothelial growth factors (VEGF) expression and down regulation of thrombospondin 1 (TSP-1) expression from the tumor cells.

Citation: Hassan M, Selimovic D, El-Khattouti A, Soell M, Ghozlan H, Haikel Y, Abdelkader O, Megahed M. Hepatitis C virus-mediated angiogenesis: Molecular mechanisms and therapeutic strategies. World J Gastroenterol 2014; 20(42): 15467-15475
URL: https://www.wjgnet.com/1007-9327/full/v20/i42/15467.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i42.15467