Extrahepatic complications to cirrhosis and portal hypertension: Haemodynamic and homeostatic aspects

doi:10.3748/wjg.v20.i42.15499

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Nov 14, 2014; 20(42): 15499-15517
Published online Nov 14, 2014. doi: 10.3748/wjg.v20.i42.15499

Table 1 Vasodilating and vasoconstricting forces involved in disturbed haemodynamics in cirrhosis (Alphabetic order)

Vasodilator systems

Adenosine

Adrenomedullin

Atrial natriuretic peptide

Bradykinin

Brain natriuretic peptide

Calcitonin gene-related peptide

Carbon monoxide

Endocannabinoids

Endothelin-3

Endotoxin

Enkephalins

Glucagon

Histamine

Hydrogen sulphide

Interleukins

Natriuretic peptide of type C

Nitric oxide

Prostacyclin (PGI2)

Substance P

Tumour necrosis factor-α

Vasoactive intestinal polypeptide

Vasoconstrictor systems

Angiotensin II

Adrenaline and noradrenaline

Endothelin-1

Neuropeptide Y

Renin-angiotensin-aldosterone system

Sympathetic nervous system

Vasopressin

Citation: Møller S, Henriksen JH, Bendtsen F. Extrahepatic complications to cirrhosis and portal hypertension: Haemodynamic and homeostatic aspects. World J Gastroenterol 2014; 20(42): 15499-15517
URL: https://www.wjgnet.com/1007-9327/full/v20/i42/15499.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i42.15499