Non-alcoholic fatty liver disease: What has changed in the treatment since the beginning?

doi:10.3748/wjg.v20.i39.14219

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 21, 2014; 20(39): 14219-14229
Published online Oct 21, 2014. doi: 10.3748/wjg.v20.i39.14219

Table 1 Common causes of secondary liver steatosis

Macrovesicular steatosis	Microvesicular steatosis
Alcohol consumption	Reye’s syndrome
Parenteral nutrition	Acute fatty liver of pregnancy
Hepatitis C	HELLP syndrome
Starvation/Malnutrition	Genetic metabolic diseases (e.g., LCAT deficiency, cholesterol ester storage disease)
Abetalipoproteinemia	Heat stroke
Lipodystrophy	Drugs (valproate, anti-retroviral drugs)
Celiac disease
Wilson’s disease
Drugs (e.g., corticosteroids, tamoxifen, amiodarone)

HELLP syndrome: Hemolysis, elevated liver enzymes and low platelets syndrome; LCAT: Lecithin-cholesterol-acyltransferase.

Citation: Baran B, Akyüz F. Non-alcoholic fatty liver disease: What has changed in the treatment since the beginning? World J Gastroenterol 2014; 20(39): 14219-14229
URL: https://www.wjgnet.com/1007-9327/full/v20/i39/14219.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i39.14219