Gene therapy in pancreatic cancer

doi:10.3748/wjg.v20.i37.13343

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Oct 7, 2014 (publication date) through Nov 23, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 7, 2014; 20(37): 13343-13368
Published online Oct 7, 2014. doi: 10.3748/wjg.v20.i37.13343

Table 3 Active and negative regulatory factors of angiogenesis

Stimulators	Inhibitors
Hypoxia	Angiostatin
Oncogenic proteins such as Ras	TSP-1
Inflammatory cytokines such as IL-8 and IL-6	Endostatin
FGF	Arrestin
TGF-β	Canstatin
HGF	MMPIs
PDGF	Somatostatin
G-CSF	Tumstatin
Angiogenin	VEGI
Leptin	Decoy receptors such as soluble VEGFR
Proliferin	Inflammatory cytokines such as IL-12

G-CSF: Granulocyte colony-stimulating factor; PDGF: Platelet-derived growth factor; HGF: Hepatocyte growth factor; TGF-β: Transforming growth factor-β; FGF: Fibroblast growth factor; TSP-1: Thrombospondin-1; MMPIs: Matrix metalloproteinases inhibitors; VEGI: Vascular endothelial growth inhibitor.

Citation: Liu SX, Xia ZS, Zhong YQ. Gene therapy in pancreatic cancer. World J Gastroenterol 2014; 20(37): 13343-13368
URL: https://www.wjgnet.com/1007-9327/full/v20/i37/13343.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i37.13343