Aspirin inhibits cell viability and mTOR downstream signaling in gastroenteropancreatic and bronchopulmonary neuroendocrine tumor cells

Figure 8 The effect of aspirin on cell cycle distribution of neuroendocrine BON1 and NCI-H727 tumor cells. Human pancreatic neuroendocrine BON1 (A) and bronchopulmonary NCI-H727 (B) cells were cultured in serum-free medium (0.2% BSA) for 24 h and then treated with the indicated concentrations of aspirin for 48 h. The mean ± SDs of 8 independently performed experiments in duplicates are shown; ^aP < 0.05, ^bP < 0.01 vs untreated control.