Establishment of chronic hepatitis C virus infection: Translational evasion of oxidative defence

doi:10.3748/wjg.v20.i11.2785

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Mar 21, 2014 (publication date) through Jul 6, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Mar 21, 2014; 20(11): 2785-2800
Published online Mar 21, 2014. doi: 10.3748/wjg.v20.i11.2785

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Figure 1 Hepatitis C virus genome. The 9.6 kb hepatitis C virus genome is flanked by highly structured 5’ untranslated region (UTR) and 3’ UTR which contain regulatory elements. The genomic sequence is translated into a polyprotein via internal ribosome entry site (IRES)-mediated translation. The polyprotein is then cleaved into individual structural and non-structural (NS) proteins by cellular signal peptidase and signal peptide peptidase and viral auto protease (NS2) and serine protease (NS3).

Citation: Chan SW. Establishment of chronic hepatitis C virus infection: Translational evasion of oxidative defence. World J Gastroenterol 2014; 20(11): 2785-2800
URL: https://www.wjgnet.com/1007-9327/full/v20/i11/2785.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i11.2785