Exogenous bone morphogenetic protein-7 reduces hepatic fibrosis in Schistosoma japonicum-infected mice via transforming growth factor-&beta;/Smad signaling

doi:10.3748/wjg.v19.i9.1405

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Mar 7, 2013; 19(9): 1405-1415
Published online Mar 7, 2013. doi: 10.3748/wjg.v19.i9.1405

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Figure 4 Representative electrophoretic bands of transforming growth factor-beta 1, alpha-smooth muscle actin, pSmad2, pSmad3 and Smad7 mRNA (reverse transcription-polymerase chain reaction). Numbers 1-3 represent group A, B and C, respectively, at 9 wk; Numbers 4-6 represent group A, B and C, respectively, at 15 wk. The 132 bp glyceraldehyde-3-phosphate dehydrogenase (GAPDH) mRNA fragment was used as an internal control. M: Marker; TGF-β1: Transforming growth factor-beta 1; α-SMA: Alpha-smooth muscle actin.

Citation: Chen BL, Peng J, Li QF, Yang M, Wang Y, Chen W. Exogenous bone morphogenetic protein-7 reduces hepatic fibrosis in Schistosoma japonicum-infected mice via transforming growth factor-β/Smad signaling. World J Gastroenterol 2013; 19(9): 1405-1415
URL: https://www.wjgnet.com/1007-9327/full/v19/i9/1405.htm
DOI: https://dx.doi.org/10.3748/wjg.v19.i9.1405