From fatty liver to fibrosis: A tale of &ldquo;second hit&rdquo;

doi:10.3748/wjg.v19.i8.1158

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 19, Issue 8

This Article

Citation of this article

Corresponding Author of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (6393)

All Articles published online

The chart showing PDF series, HTML series, Figures (1-2) series.

Item

Count

PDF

685

HTML

5524

Figures (1-2)

184

Sum=6393

Feb 28, 2013 (publication date) through Jul 21, 2024

Times Cited of This Article

Times Cited (123)

Journal Information of This Article

Publication Name

World Journal of Gastroenterology

ISSN

1007-9327

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Topic Highlight

World J Gastroenterol. Feb 28, 2013; 19(8): 1158-1165
Published online Feb 28, 2013. doi: 10.3748/wjg.v19.i8.1158

Full Size Figure Download Figure

Figure 2 Logical and attractive hypothesis is that oxidative stress in TG-loaded hepatocytes is the cause of sustained injury with consequent NASH, fibrosis and cirrhosis. HSP: Heat shock proteins.

Citation: Basaranoglu M, Basaranoglu G, Sentürk H. From fatty liver to fibrosis: A tale of “second hit”. World J Gastroenterol 2013; 19(8): 1158-1165
URL: https://www.wjgnet.com/1007-9327/full/v19/i8/1158.htm
DOI: https://dx.doi.org/10.3748/wjg.v19.i8.1158