Abnormal DNA-PKcs and Ku 70/80 expression may promote malignant pathological processes in gastric carcinoma

doi:10.3748/wjg.v19.i40.6894

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Oct 28, 2013 (publication date) through Aug 1, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 28, 2013; 19(40): 6894-6901
Published online Oct 28, 2013. doi: 10.3748/wjg.v19.i40.6894

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Figure 3 A model depicting the effect of Helicobacter pylori infection on the DNA-dependent protein kinase and Ku 70/80-mediated non-homologous end joining repair pathway in gastric epithelial cells that might ultimately promote carcinogenesis. ATM: Ataxia telangiectasia mutated; DNA-PKcs: DNA-dependent protein kinase.

Citation: Li W, Xie C, Yang Z, Chen J, Lu NH. Abnormal DNA-PKcs and Ku 70/80 expression may promote malignant pathological processes in gastric carcinoma. World J Gastroenterol 2013; 19(40): 6894-6901
URL: https://www.wjgnet.com/1007-9327/full/v19/i40/6894.htm
DOI: https://dx.doi.org/10.3748/wjg.v19.i40.6894