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©2013 Baishideng Publishing Group Co.
World J Gastroenterol. Jun 7, 2013; 19(21): 3226-3240
Published online Jun 7, 2013. doi: 10.3748/wjg.v19.i21.3226
Published online Jun 7, 2013. doi: 10.3748/wjg.v19.i21.3226
Figure 4 AGS cell cycle changes before and after trichostatin A exposure by flow cytometry.
The AGS cell cycle ratio before TSA treatment was: %G1 = 26, %S = 53.5, %G2 = 17.7, and the AGS cell cycle ratio after 0.25 μmol/L trichostatin A treatment was: %G1 = 44.6, %S = 20.9, %G2 = 31.3. Cycle arrest occurred in G0/G1, G2/M phases, especially in G0/G1 phase. TSA: Trichostatin A.
- Citation: Wang YG, Wang N, Li GM, Fang WL, Wei J, Ma JL, Wang T, Shi M. Mechanisms of trichostatin A inhibiting AGS proliferation and identification of lysine-acetylated proteins. World J Gastroenterol 2013; 19(21): 3226-3240
- URL: https://www.wjgnet.com/1007-9327/full/v19/i21/3226.htm
- DOI: https://dx.doi.org/10.3748/wjg.v19.i21.3226