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©2013 Baishideng Publishing Group Co.
World J Gastroenterol. Apr 21, 2013; 19(15): 2419-2424
Published online Apr 21, 2013. doi: 10.3748/wjg.v19.i15.2419
Published online Apr 21, 2013. doi: 10.3748/wjg.v19.i15.2419
Table 2 Incidence of inflammation, intestinal metaplasia, intestinal metaplasia with dysplasia, and adenocarcinoma in the esophagus and forestomach 40 wk after surgery n (%)
| Inflammation | Intestinal metaplasia | Intestinal metaplasia with dysplasia | Adenocarcinoma | |
| Control esophagus (n = 29) | 2 (6.8) | 1 (3.4) | 0 | 0 |
| Model esophagus (n = 25) | 24 (96) | 22 (88) | 5 (20) | 2 (8) |
| Control forestomach (n = 29) | 2 (6.8) | 1 (3.4) | 0 | 0 |
| Model forestomach (n = 25) | 25 (100) | 2 (8) | 0 | 0 |
- Citation: Cheng P, Li JS, Zhang LF, Chen YZ, Gong J. Exposure to gastric juice may not cause adenocarcinogenesis of the esophagus. World J Gastroenterol 2013; 19(15): 2419-2424
- URL: https://www.wjgnet.com/1007-9327/full/v19/i15/2419.htm
- DOI: https://dx.doi.org/10.3748/wjg.v19.i15.2419