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World J Gastroenterol. Mar 28, 2013; 19(12): 2000-2004
Published online Mar 28, 2013. doi: 10.3748/wjg.v19.i12.2000
Published online Mar 28, 2013. doi: 10.3748/wjg.v19.i12.2000
Figure 3 Histopathology changes after treatment.
A: Histopathological examination revealed that the lesion had chronic mucosal inflammation with acute inflammation and a small amount of Helicobacter pylori (H. pylori) infection. The mitotic phase was obvious and lymphoma was widespread [hematoxylin and eosin (HE), ×400, July 2, 2011]; B: Histopathological examination revealed chronic mucosal inflammation with acute inflammation and a only a small amount of H. pylori infection. The mitotic phase was 4/10 high power field (HE, ×400, August 17, 2011); C: Immunohistochemical staining showed tissue cell-like cells with S-100 (×400, August 17, 2011); D: Immunohistochemical staining was repeated by the Beijing Cancer Hospital and showed that staining for CD1a was positive for focal lesions (×400, August 25, 2011); E: Histopathological examination showed chronic mucosal inflammation with lymphoid tissue hyperplasia in the lamina propria (HE, ×400, October 14, 2011); F: Histopathological examination revealed that chronic mucosal inflammation with acute inflammation (HE, ×10 , May 8, 2012).
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Citation: Li TT, Qiu F, Wang ZQ, Sun L, Wan J. Rare case of
Helicobacter pylori -related gastric ulcer: Malignancy or pseudomorphism? World J Gastroenterol 2013; 19(12): 2000-2004 - URL: https://www.wjgnet.com/1007-9327/full/v19/i12/2000.htm
- DOI: https://dx.doi.org/10.3748/wjg.v19.i12.2000