Dietary copper triggers onset of fulminant hepatitis in the Long-Evans cinnamon rat model

doi:10.3748/wjg.v18.i39.5542

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Oct 21, 2012 (publication date) through Feb 12, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 21, 2012; 18(39): 5542-5550
Published online Oct 21, 2012. doi: 10.3748/wjg.v18.i39.5542

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Figure 5 Ceruloplasmin oxidase is independent of high copper treatment. Ceruloplasmin (CP) oxidase was determined in Long-Evans cinnamon rats (-/-) that received high copper (hCu) or reduced copper (rCu). Sera (n = 6) were obtained from animals after an early or late hCu regimen. As control, values obtained from Long-Evans agouti rats (wt) (n = 3) are shown. Data are expressed as mean ± SE.

Citation: Siaj R, Sauer V, Stöppeler S, Spiegel HU, Köhler G, Zibert A, Schmidt HH. Dietary copper triggers onset of fulminant hepatitis in the Long-Evans cinnamon rat model. World J Gastroenterol 2012; 18(39): 5542-5550
URL: https://www.wjgnet.com/1007-9327/full/v18/i39/5542.htm
DOI: https://dx.doi.org/10.3748/wjg.v18.i39.5542