Stem cell factor-mediated wild-type KIT receptor activation is critical for gastrointestinal stromal tumor cell growth

doi:10.3748/wjg.v18.i23.2929

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Jun 21, 2012 (publication date) through Nov 3, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jun 21, 2012; 18(23): 2929-2937
Published online Jun 21, 2012. doi: 10.3748/wjg.v18.i23.2929

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Figure 2 The expression of stem cell factor in primary gastrointestinal stromal tumors. Sections of gastrointestinal stromal tumors (GISTs) (n = 51) were immunostained with stem cell factor (SCF), KIT (CD117), pHH3 and Ki-67 antibodies (magnification, ×200). Apoptosis was assessed in situ by terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end labeling (TUNEL) staining (magnification, ×200). Representative images are shown. Bar = 50 μm.

Citation: Bai CG, Hou XW, Wang F, Qiu C, Zhu Y, Huang L, Zhao J, Xu JJ, Ma DL. Stem cell factor-mediated wild-type KIT receptor activation is critical for gastrointestinal stromal tumor cell growth. World J Gastroenterol 2012; 18(23): 2929-2937
URL: https://www.wjgnet.com/1007-9327/full/v18/i23/2929.htm
DOI: https://dx.doi.org/10.3748/wjg.v18.i23.2929