Original Article
Copyright ©2012 Baishideng Publishing Group Co.
World J Gastroenterol. Jun 21, 2012; 18(23): 2914-2928
Published online Jun 21, 2012. doi: 10.3748/wjg.v18.i23.2914
Figure 4
Figure 4 Schematic drawing summarizes the major findings that might associate with pathophysiological changes in rat nervous system caused by trinitrobenzene sulfonic acid-induced colitis. TNBS: Trinitrobenzene sulfonic acid; DRG: Dorsal root ganglia; Hnrnpa2b1: Heterogeneous nuclear ribonucleoproteins A2/B1; Gstp1: Glutathione S-transferase P; Prdx1: Peroxiredoxin-1; Sod2: Superoxide dismutase; Lmna: Lamin C2; Aldoa: Aldolase A; Aldoc: Aldolase C; Ckmt1: Creatine kinase, mitochondrial 1, ubiquitous; Ckm: Creatine kinase M-type; Ndufv2: NADH dehydrogenase (ubiquinone) flavoprotein 2; Ndufb10: NADH dehydrogenase (ubiquinone) 1 β subcomplex 10; Oxct1 succinyl-CoA: 3-ketoacid-coenzyme A transferase 1; Fh1: Fumarate hydratase; Mdh2: Malate dehydrogenase; Psat1: Phosphoserine aminotransferase; Cap1: Cyclase-associated protein 1; Kctd12: Potassium channel tetramerisation domain containing protein 12; Vdac2: Voltage-dependent anion-selective channel protein 2; Ywhae: The 14-3-3 protein epsilon; Tst: Thiosulfate sulfurtransferase; Stip1: Stress-induced phosphoprotein 1; Hspa8: heat shock cognate 71 kDa protein.