HBx activates FasL and mediates HepG2 cell apoptosis through MLK3-MKK7-JNKs signal module

doi:10.3748/wjg.v18.i13.1485

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Apr 7, 2012 (publication date) through Jul 25, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Apr 7, 2012; 18(13): 1485-1495
Published online Apr 7, 2012. doi: 10.3748/wjg.v18.i13.1485

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Figure 4 Upregulation of Fas/FasL signaling pathway-related protein expression by hepatitis B virus X protein in a time-dependent manner. HepG2 cells were transfected with pcDNA3.1-X and incubated for various time periods as indicated. The levels of Fas, FasL mRNA (A) and proteins (B) were determined by reverse transcription polymerase chain reaction and Western blotting analysis. Data are expressed as mean ± SD (n = 3).

Citation: Tang RX, Kong FY, Fan BF, Liu XM, You HJ, Zhang P, Zheng KY. HBx activates FasL and mediates HepG2 cell apoptosis through MLK3-MKK7-JNKs signal module. World J Gastroenterol 2012; 18(13): 1485-1495
URL: https://www.wjgnet.com/1007-9327/full/v18/i13/1485.htm
DOI: https://dx.doi.org/10.3748/wjg.v18.i13.1485