Epidemiological aspects of Budd-Chiari in Egyptian patients: A single-center study

doi:10.3748/wjg.v17.i42.4704

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Nov 14, 2011 (publication date) through Nov 27, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Nov 14, 2011; 17(42): 4704-4710
Published online Nov 14, 2011. doi: 10.3748/wjg.v17.i42.4704

Table 2 The primary etiologies of Budd-Chiari syndrome (n = 94)

Etiology		n (%)
FVLM (64 tested)	Homozygous	10 (15.6)
FVLM (64 tested)	Heterozygous	24 (37.5)
MTHFR (60 tested)	Homozygous	8 (13.3)
MTHFR (60 tested)	Heterozygous	23 (38.3)
PGM (60 tested)	Homozygous	1 (1.7)
PGM (60 tested)	Heterozygous	2 (3.3)
JAK2 (MPD) (62 tested)	+	18 (29)
Primary APA	+	16 (17)
Secondary APA	+	11 (11.7)
Behçet’s disease	+	12 (12.8)
Protein C deficiency	+	4 (4.3)
Antithrombin III deficiency	+	4 (4.3)
Protein S deficiency	+	1 (1.1)
PNH	+	2 (2.1)
Hormonal therapy (58 females)	+	9 (15.5)
Pregnancy-related (58 females)	+	10 (17.2)

FVLM: Factor V Leiden mutation; MTHFR: Methylene tetrahydrofolate reductase; PGM: Prothrombin gene mutation; JAK2: Janus tyrosine kinase-2; MPD: Myeloproliferative disorder; APA: Antiphospholipid antibody syndrome; PNH: Paroxysmal nocturnal hemoglobinuria.

Citation: Sakr M, Barakat E, Abdelhakam S, Dabbous H, Yousuf S, Shaker M, Eldorry A. Epidemiological aspects of Budd-Chiari in Egyptian patients: A single-center study. World J Gastroenterol 2011; 17(42): 4704-4710
URL: https://www.wjgnet.com/1007-9327/full/v17/i42/4704.htm
DOI: https://dx.doi.org/10.3748/wjg.v17.i42.4704