Casticin-induced apoptosis involves death receptor 5 upregulation in hepatocellular carcinoma cells

doi:10.3748/wjg.v17.i38.4298

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 17, Issue 38

This Article

Citation of this article

Corresponding Author of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (4769)

All Articles published online

The chart showing PDF series, HTML series, Figures (1-7) series.

Item

Count

PDF

404

HTML

4175

Figures (1-7)

190

Sum=4769

Oct 14, 2011 (publication date) through Jul 25, 2024

Times Cited of This Article

Times Cited (36)

Journal Information of This Article

Publication Name

World Journal of Gastroenterology

ISSN

1007-9327

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Original Article

World J Gastroenterol. Oct 14, 2011; 17(38): 4298-4307
Published online Oct 14, 2011. doi: 10.3748/wjg.v17.i38.4298

Full Size Figure Download Figure

Figure 6 Effects of casticin on intracellular glutathione content (A, B and D, E) and the percentage sub-G1 cell population (C and F) in PLC/PRF/5 and Hep G2 cells (mean ± SD, n = 3). ^aP < 0.05 vs treatment with dimethyl-sulfoxide (DMSO) or 0 h; ^bP < 0.05 vs treatment with 30 μmol/L casticin alone or treatment with 10 μmol/L casticin for 1 h. GSH: Glutathione; BHA: Butylated hydroxyanisole; NAC: N-acetyl-cysteine.

Citation: Yang J, Yang Y, Tian L, Sheng XF, Liu F, Cao JG. Casticin-induced apoptosis involves death receptor 5 upregulation in hepatocellular carcinoma cells. World J Gastroenterol 2011; 17(38): 4298-4307
URL: https://www.wjgnet.com/1007-9327/full/v17/i38/4298.htm
DOI: https://dx.doi.org/10.3748/wjg.v17.i38.4298