Casticin-induced apoptosis involves death receptor 5 upregulation in hepatocellular carcinoma cells

doi:10.3748/wjg.v17.i38.4298

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Oct 14, 2011 (publication date) through Nov 8, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 14, 2011; 17(38): 4298-4307
Published online Oct 14, 2011. doi: 10.3748/wjg.v17.i38.4298

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Figure 6 Effects of casticin on intracellular glutathione content (A, B and D, E) and the percentage sub-G1 cell population (C and F) in PLC/PRF/5 and Hep G2 cells (mean ± SD, n = 3). ^aP < 0.05 vs treatment with dimethyl-sulfoxide (DMSO) or 0 h; ^bP < 0.05 vs treatment with 30 μmol/L casticin alone or treatment with 10 μmol/L casticin for 1 h. GSH: Glutathione; BHA: Butylated hydroxyanisole; NAC: N-acetyl-cysteine.

Citation: Yang J, Yang Y, Tian L, Sheng XF, Liu F, Cao JG. Casticin-induced apoptosis involves death receptor 5 upregulation in hepatocellular carcinoma cells. World J Gastroenterol 2011; 17(38): 4298-4307
URL: https://www.wjgnet.com/1007-9327/full/v17/i38/4298.htm
DOI: https://dx.doi.org/10.3748/wjg.v17.i38.4298