NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn&rsquo;s disease

doi:10.3748/wjg.v17.i23.2829

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Jun 21, 2011 (publication date) through Nov 2, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jun 21, 2011; 17(23): 2829-2837
Published online Jun 21, 2011. doi: 10.3748/wjg.v17.i23.2829

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Figure 2 Effect of NOD2 variants on mycobacterium avium subspecies paratuberculosis numbers. Time course of mycobacterium avium subspecies paratuberculosis (MAP) growth in the context of NOD2 genetic variation. Monocytes were derived from subjects homozygous for the ATG16L1 1138G allele and with/without NOD2 genetic variants (n = 6 for each group). MAP growth is expressed as colony forming units (cfu).

Citation: Glubb DM, Gearry RB, Barclay ML, Roberts RL, Pearson J, Keenan JI, McKenzie J, Bentley RW. NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn’s disease. World J Gastroenterol 2011; 17(23): 2829-2837
URL: https://www.wjgnet.com/1007-9327/full/v17/i23/2829.htm
DOI: https://dx.doi.org/10.3748/wjg.v17.i23.2829