NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn&rsquo;s disease

doi:10.3748/wjg.v17.i23.2829

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jun 21, 2011; 17(23): 2829-2837
Published online Jun 21, 2011. doi: 10.3748/wjg.v17.i23.2829

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Figure 1 Effect of ATG16L1 T300A variant on mycobacterium avium subspecies paratuberculosis numbers. Time course of mycobacterium avium subspecies paratuberculosis (MAP) growth in the context of the ATG16L1 1138 G > A variant. Monocytes were derived from subjects homozygous for either ATG16L1 allele and carrying NOD2 wild-type alleles (n = 6 for each group). MAP growth is expressed as colony forming units (cfu).

Citation: Glubb DM, Gearry RB, Barclay ML, Roberts RL, Pearson J, Keenan JI, McKenzie J, Bentley RW. NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn’s disease. World J Gastroenterol 2011; 17(23): 2829-2837
URL: https://www.wjgnet.com/1007-9327/full/v17/i23/2829.htm
DOI: https://dx.doi.org/10.3748/wjg.v17.i23.2829