Role of nitric oxide in hepatic ischemia-reperfusion injury

doi:10.3748/wjg.v16.i48.6079

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Dec 28, 2010 (publication date) through Jan 30, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Dec 28, 2010; 16(48): 6079-6086
Published online Dec 28, 2010. doi: 10.3748/wjg.v16.i48.6079

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Figure 1 Multifaceted hepatic ischemia-reperfusion injury. Kupffer and endothelial cells produce cytokines and chemokines, recruiting neutrophils that further accentuate injury. EC: Endothelial cell; KC: Kupffer cell; ATP: Adenosine triphosphate; TNF: Tumor necrosis factor; IL: Interleukin; ICAM: Intercellular adhesion molecule; VCAM: Vascular adhesion molecule; PAF: Platelet activation factor; LTB4: Leukotriene B4; GMS-CSF: Granulocyte macrophage colony stimulating factor; INF: Interferon; ROS: Reactive oxygen species (Courtesy of Dr. Joan Rosello-Catafau, Barcelona, Spain).

Citation: Siriussawakul A, Zaky A, Lang JD. Role of nitric oxide in hepatic ischemia-reperfusion injury. World J Gastroenterol 2010; 16(48): 6079-6086
URL: https://www.wjgnet.com/1007-9327/full/v16/i48/6079.htm
DOI: https://dx.doi.org/10.3748/wjg.v16.i48.6079