Acute lung injury and ARDS in acute pancreatitis: Mechanisms and potential intervention

doi:10.3748/wjg.v16.i17.2094

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May 7, 2010 (publication date) through Feb 18, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. May 7, 2010; 16(17): 2094-2099
Published online May 7, 2010. doi: 10.3748/wjg.v16.i17.2094

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Figure 3 Acute pancreatitis-associated acute lung injury (ALI) - potential mechanisms including endothelial barrier dysfunction. Several adhesion molecules [selectins, intercellular adhesion molecule-1 (ICAM-1), platelet endothelial cell adhesion molecule-1 (PECAM-1) among others] involved in the extravasation of not at least polymorphonuclear neutrophils (PMNs). Tissue injury by not at least these PMNs.

Citation: Zhou MT, Chen CS, Chen BC, Zhang QY, Andersson R. Acute lung injury and ARDS in acute pancreatitis: Mechanisms and potential intervention. World J Gastroenterol 2010; 16(17): 2094-2099
URL: https://www.wjgnet.com/1007-9327/full/v16/i17/2094.htm
DOI: https://dx.doi.org/10.3748/wjg.v16.i17.2094