Mechanism and pathobiologic implications of CHFR promoter methylation in gastric carcinoma

doi:10.3748/wjg.14.5000

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Aug 28, 2008 (publication date) through Oct 2, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Gastric Cancer

World J Gastroenterol. Aug 28, 2008; 14(32): 5000-5007
Published online Aug 28, 2008. doi: 10.3748/wjg.14.5000

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Figure 1 Representative results of MSPCR in human GC tissue samples. Lanes U and M: Products derived from unmethylated and methylated alleles, respectively. Methylation of the CHFR gene was detected in 16 and 6 poorly-differentiated adenocarcinoma tissue samples and 1 mucinous adenocarcinoma tissue sample, while unmethylation of the CHFR gene was detected in 7 well-differentiated adenocarcinoma tissue samples. PUC18: Marker; N: Normal gastric mucosa corresponding to tumors; T: Gastric cancer.

Citation: Gao YJ, Xin Y, Zhang JJ, Zhou J. Mechanism and pathobiologic implications of CHFR promoter methylation in gastric carcinoma. World J Gastroenterol 2008; 14(32): 5000-5007
URL: https://www.wjgnet.com/1007-9327/full/v14/i32/5000.htm
DOI: https://dx.doi.org/10.3748/wjg.14.5000