JAK-STAT pathway in carcinogenesis: Is it relevant to cholangiocarcinoma progression

doi:10.3748/wjg.v13.i48.6478

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Dec 28, 2007 (publication date) through Dec 4, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Dec 28, 2007; 13(48): 6478-6491
Published online Dec 28, 2007. doi: 10.3748/wjg.v13.i48.6478

Table 4 Nuclear prolactin receptor expression in rat liver transplanted RS-1 cholangiocarcinoma cells and adjacent hepatocytes as compared with normal cells

	Nuclear PrlR-positive immunoreactivity (arbitrary units), medians (upper and lower quartiles)
	Normal cholangiocytes	RS-1 cholangiocarcinoma cells	Normal hepatocytes	Tumor adjacent hepatocytes
Males	47 (0; 104)	191^b (119; 288)	40 (26; 90)	308^b (227; 381)
Females	113 (47; 194)	408^b (256; 555)	59 (41; 101)	415^b (266; 581)

^bP < 0.001 as compared with correspondent intact group, Mann-Witney U-test.

Citation: Smirnova OV, Ostroukhova TY, Bogorad RL. JAK-STAT pathway in carcinogenesis: Is it relevant to cholangiocarcinoma progression. World J Gastroenterol 2007; 13(48): 6478-6491
URL: https://www.wjgnet.com/1007-9327/full/v13/i48/6478.htm
DOI: https://dx.doi.org/10.3748/wjg.v13.i48.6478