Ethanol induced mitochondria injury and permeability transition pore opening: Role of mitochondria in alcoholic liver disease

doi:10.3748/wjg.v13.i16.2352

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Apr 28, 2007 (publication date) through Jan 19, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Apr 28, 2007; 13(16): 2352-2356
Published online Apr 28, 2007. doi: 10.3748/wjg.v13.i16.2352

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Figure 1 Mitochondria appearance under electron microscope (EM × 6000); A: Mitochondria in normal group; B: Mitochondria in model group. M: mitochondria, G: glycogen, N nucleus, ER: endoplasmic reticulum, LD: lipid droplet. The long arrow shows abnormally distributed chromatin in nuclei, the short one is megamitochondrion and the arrow head is U-type mitochondria.

Citation: Yan M, Zhu P, Liu HM, Zhang HT, Liu L. Ethanol induced mitochondria injury and permeability transition pore opening: Role of mitochondria in alcoholic liver disease. World J Gastroenterol 2007; 13(16): 2352-2356
URL: https://www.wjgnet.com/1007-9327/full/v13/i16/2352.htm
DOI: https://dx.doi.org/10.3748/wjg.v13.i16.2352