Roles of the MEK1/2 and AKT pathways in CXCL12/CXCR4 induced cholangiocarcinoma cell invasion

doi:10.3748/wjg.v13.i10.1561

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Mar 14, 2007 (publication date) through Mar 7, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Mar 14, 2007; 13(10): 1561-1568
Published online Mar 14, 2007. doi: 10.3748/wjg.v13.i10.1561

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Figure 9 Effect of CXCL12 on the polymerization of actin cytoskeleton. Cholangiocarcinoma cells were pre-treated with vehicle, AMD3100, U0126 or LY294002 and incubated for 6 h in medium containing 0 or 100 ng/mL CXCL12. The cells were stained with Alexa Fluor 488 Phalloidin to visualize actin cytoskeleton under a Confocal laser scanning microscope (Olympus SV1000).

Citation: Leelawat K, Leelawat S, Narong S, Hongeng S. Roles of the MEK1/2 and AKT pathways in CXCL12/CXCR4 induced cholangiocarcinoma cell invasion. World J Gastroenterol 2007; 13(10): 1561-1568
URL: https://www.wjgnet.com/1007-9327/full/v13/i10/1561.htm
DOI: https://dx.doi.org/10.3748/wjg.v13.i10.1561