Copyright
©2006 Baishideng Publishing Group Co.
World J Gastroenterol. Nov 21, 2006; 12(43): 7029-7032
Published online Nov 21, 2006. doi: 10.3748/wjg.v12.i43.7029
Published online Nov 21, 2006. doi: 10.3748/wjg.v12.i43.7029
Normal | CA | ||||
Hpylori (+) | H pylori (-) | H pylori (+) | Hpylori (-) | ||
n | 11 (%) | 14 (%) | 11 (%) | 14 (%) | P |
Overall | 100.0 | 14.3 | 100.0 | 92.9 | < 0.0001 |
116 kDa | 100.0 | 14.3 | 90.9 | 71.4 | 0.006 |
89 kDa | 44.4 | 0.0 | 60.0 | 35.7 | 0.04 |
35 kDa | 100.0 | 0.0 | 100.0 | 57.1 | 0.002 |
30 kDa | 100.0 | 7.14 | 81.8 | 75.1 | 0.01 |
26.5 kDa | 90.9 | 14.3 | 72.7 | 71.4 | 0.006 |
19.5 kDa | 72.7 | 0.0 | 40.0 | 14.3 | 0.482 |
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Citation: Yang KC, Chu A, Liao CS, Lin YM, Wang GM. Evaluation of the role of
H pylori infection in pathogenesis of gastric cancer by immunoblot assay. World J Gastroenterol 2006; 12(43): 7029-7032 - URL: https://www.wjgnet.com/1007-9327/full/v12/i43/7029.htm
- DOI: https://dx.doi.org/10.3748/wjg.v12.i43.7029