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©2006 Baishideng Publishing Group Co.
World J Gastroenterol. Aug 7, 2006; 12(29): 4721-4726
Published online Aug 7, 2006. doi: 10.3748/wjg.v12.i29.4721
Published online Aug 7, 2006. doi: 10.3748/wjg.v12.i29.4721
Patients(n = 252) | Patients(Only genotype 3)(n = 187) | Healthycontrols(n = 408) | |
Age (yr) | |||
Mean ± SD | 40 ± 14 | 40 ± 14 | 33 ± 7 |
Male: Female | 164:88 | 118:69 | 297:111 |
ALT (IU/L) | |||
Mean | 112.3 ± 68.4 | 116 ± 70.1 | 29 ± 2 |
Range | (32-330) | (32-330) | |
S. Albumin (g/L) | 36 ± 4 | 34 ± 5 | 40 ± 3 |
HCV genotype (n = 209) | |||
I | 17 (8.1%) | - | |
II | 1 (0.5%) | ||
III | 187 (89.4%) | - | |
IV | 4 (1.5%) | - | |
Liver Biopsy | |||
Fibrosis score | 2 ± 1 | 2 ± 1.2 | - |
HAI | 5.5 ± 2.4 | 5.6 ± 2.5 | - |
Mode of Acquisition of HCV | |||
Blood transfusion | 86 | - | |
Needle stick injury | 54 | - | |
Intravenous drug use | 10 | - | |
Medical procedure | 45 | - | |
Unclear | 57 |
- Citation: Goyal A, Suneetha P, Kumar G, Shukla DK, Arora N, Sarin SK. CCR5Δ32 mutation does not influence the susceptibility to HCV infection, severity of liver disease and response to therapy in patients with chronic hepatitis C. World J Gastroenterol 2006; 12(29): 4721-4726
- URL: https://www.wjgnet.com/1007-9327/full/v12/i29/4721.htm
- DOI: https://dx.doi.org/10.3748/wjg.v12.i29.4721