Gut hormones, and short bowel syndrome: The enigmatic role of glucagon-like peptide-2 in the regulation of intestinal adaptation

doi:10.3748/wjg.v12.i26.4117

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Jul 14, 2006 (publication date) through Dec 23, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jul 14, 2006; 12(26): 4117-4129
Published online Jul 14, 2006. doi: 10.3748/wjg.v12.i26.4117

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Figure 5 Neural or humoral mechanism involvement in the early release of GLP-2. The delivery of a meal significantly increases serum GLP-2 levels in rats with discontinuous small bowel (ANOVA, P < 0.007). The disruption of the continuity of the bowel (stoma creation) did not inhibit the initial nutrient-stimulated GLP-2 response. Thus, GLP-2 production is increased at least in part by neural and/or hormonal mechanisms. Serum GLP-2 levels reported as ng/mL.

Citation: Martin G, Beck P, Sigalet D. Gut hormones, and short bowel syndrome: The enigmatic role of glucagon-like peptide-2 in the regulation of intestinal adaptation. World J Gastroenterol 2006; 12(26): 4117-4129
URL: https://www.wjgnet.com/1007-9327/full/v12/i26/4117.htm
DOI: https://dx.doi.org/10.3748/wjg.v12.i26.4117