Cis-hydroxyproline-induced inhibition of pancreatic cancer cell growth is mediated by endoplasmic reticulum stress

doi:10.3748/wjg.v12.i10.1569

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Mar 14, 2006 (publication date) through Dec 2, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Mar 14, 2006; 12(10): 1569-1576
Published online Mar 14, 2006. doi: 10.3748/wjg.v12.i10.1569

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Figure 4 Immunocytochemical localisation of FAK. DSL6A cells grown on glass cover slips were cultured for 24 h without (A) or with 10 mmol/L CHP (B). Cell staining was performed by incubation with a mouse anti-FAK specific antibody. Binding of the primary antibody was detected by an AlexaFluorTM-labelled anti-mouse antibody. Arrows: focal adhesions; Bar: 10 μm.

Citation: Mueller C, Emmrich J, Jaster R, Braun D, Liebe S, Sparmann G. Cis-hydroxyproline-induced inhibition of pancreatic cancer cell growth is mediated by endoplasmic reticulum stress. World J Gastroenterol 2006; 12(10): 1569-1576
URL: https://www.wjgnet.com/1007-9327/full/v12/i10/1569.htm
DOI: https://dx.doi.org/10.3748/wjg.v12.i10.1569