Mutations outside the YMDD motif in the P protein can also cause DHBV resistant to Lamivudine

doi:10.3748/wjg.v11.i27.4261

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Jul 21, 2005 (publication date) through Nov 4, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jul 21, 2005; 11(27): 4261-4267
Published online Jul 21, 2005. doi: 10.3748/wjg.v11.i27.4261

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Figure 4 Electrophoresis graph of the amplification of DHBV complete genome and enzyme cutting of the recombinant. Lanes 1 and 7: λ-EcoT14I digest marker; lane 2: the LAPCR product of DHBV complete genome; lane 3: the cloning product of DHBV complete genome; lane 4: EcoRI enzyme cutting product of DHBV complete genome recombinants; lane 5: loop product of pMD18-T vector; lane 6: EcoRI enzyme cutting product of loop product of pMD18-T vector.

Citation: He JY, Zhu YT, Yang RY, Feng LL, Guo XB, Zhang FX, Chen HS. Mutations outside the YMDD motif in the P protein can also cause DHBV resistant to Lamivudine. World J Gastroenterol 2005; 11(27): 4261-4267
URL: https://www.wjgnet.com/1007-9327/full/v11/i27/4261.htm
DOI: https://dx.doi.org/10.3748/wjg.v11.i27.4261