Neither gastric topological distribution nor principle virulence genes of Helicobacter pylori contributes to clinical outcomes

doi:10.3748/wjg.v10.i22.3274

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Nov 15, 2004 (publication date) through Jul 25, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

H Pylori

World J Gastroenterol. Nov 15, 2004; 10(22): 3274-3277
Published online Nov 15, 2004. doi: 10.3748/wjg.v10.i22.3274

Table 3 Anatomical location of the 134 H pylori isolates and their virulence genes

	Antrum (%) n = 43	Body/Corpus (%) n = 47	Cardia(%) n = 44
Clinical Diagnosis
PUD	12 (33)	15 (42)	12 (33)
GERD	13 (26)	15 (30)	14 (28)
NUD	18 (25)	17 (24)	18 (26)
Genotype
26kDa	43 (100)	47 (100)	44 (100)
ureC	43 (100)	47 (100)	44 (100)
cagA	35 (81)	35 (74)	33 (75)
vacA	37 (86)	38 (81)	35 (80)
iceA1	25 (58)	25 (53)	26 (59)
iceA2	10 (23)	12 (26)	9 (20)
babA2	18 (42)	20 (38)	16 (36)

PUD: Peptic ulcer disease; GERD: Gastroesophageal reflux disease; NUD: Non-ulcer dyspepsia. All test values were calculated with respect to gastric antrum and none were significant.

Citation: Ho YW, Ho KY, Ascencio F, Ho B. Neither gastric topological distribution nor principle virulence genes of Helicobacter pylori contributes to clinical outcomes. World J Gastroenterol 2004; 10(22): 3274-3277
URL: https://www.wjgnet.com/1007-9327/full/v10/i22/3274.htm
DOI: https://dx.doi.org/10.3748/wjg.v10.i22.3274