Potential role of p53 mutation in chemical hepatocarcinogenesis of rats

doi:10.3748/wjg.v10.i1.46

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Jan 1, 2004 (publication date) through Nov 27, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jan 1, 2004; 10(1): 46-52
Published online Jan 1, 2004. doi: 10.3748/wjg.v10.i1.46

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Figure 3 Quantitative analysis of p53 mRNA. After rats were treated with 3’-Me-DAB for 6, 12 and 24 weeks, the expressions of p53 mRNA were markedly higher in prehepatocarcinoma than in hepatocarcinoma and adjacent normal tissue, P < 0.001. The time course of p53 mRNA showed that mRNA levels peaked at week 6, and gradually decreased to minimum at week 24. Normal = adjacent normal tissue, Precar = prehepatocarcinoma, Car = hepatocarcinoma.

Citation: Deng WG, Fu Y, Li YL, Sugiyama T. Potential role of p53 mutation in chemical hepatocarcinogenesis of rats. World J Gastroenterol 2004; 10(1): 46-52
URL: https://www.wjgnet.com/1007-9327/full/v10/i1/46.htm
DOI: https://dx.doi.org/10.3748/wjg.v10.i1.46