Potential role of p53 mutation in chemical hepatocarcinogenesis of rats

doi:10.3748/wjg.v10.i1.46

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 10, Issue 1

This Article

Citation of this article

Corresponding Author of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (2872)

All Articles published online

The chart showing PDF series, HTML series, Figures (1-7) series, Tables (1-1) series.

Item

Count

PDF

304

HTML

2160

Figures (1-7)

241

Tables (1-1)

167

Sum=2872

Jan 1, 2004 (publication date) through Aug 1, 2024

Times Cited of This Article

Times Cited (6)

Journal Information of This Article

Publication Name

World Journal of Gastroenterology

ISSN

1007-9327

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Liver Cancer

World J Gastroenterol. Jan 1, 2004; 10(1): 46-52
Published online Jan 1, 2004. doi: 10.3748/wjg.v10.i1.46

Full Size Figure Download Figure

Figure 3 Quantitative analysis of p53 mRNA. After rats were treated with 3’-Me-DAB for 6, 12 and 24 weeks, the expressions of p53 mRNA were markedly higher in prehepatocarcinoma than in hepatocarcinoma and adjacent normal tissue, P < 0.001. The time course of p53 mRNA showed that mRNA levels peaked at week 6, and gradually decreased to minimum at week 24. Normal = adjacent normal tissue, Precar = prehepatocarcinoma, Car = hepatocarcinoma.

Citation: Deng WG, Fu Y, Li YL, Sugiyama T. Potential role of p53 mutation in chemical hepatocarcinogenesis of rats. World J Gastroenterol 2004; 10(1): 46-52
URL: https://www.wjgnet.com/1007-9327/full/v10/i1/46.htm
DOI: https://dx.doi.org/10.3748/wjg.v10.i1.46