Pathogenesis and significance of hepatitis C virus steatosis: An update on survival strategy of a successful pathogen

doi:10.3748/wjg.v20.i23.7089

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 20, Issue 23

This Article

Academic Content and Language Evaluation of This Article

Citation of this article

Corresponding Author of This Article

Research Domain of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (10290)

All Articles published online

The chart showing PDF series, WORD series, HTML series, Figures (1-3) series, Tables (1-4) series.

Item

Count

PDF

798

WORD

314

HTML

6411

Figures (1-3)

157

Tables (1-4)

173

Sum=7853

Publishing Process of This Article

The chart showing Browse series, Download series.

Item

Count

Browse

1237

Download

1200

Sum=2437

Jun 21, 2014 (publication date) through Jun 4, 2024

Times Cited of This Article

Times Cited (74)

Journal Information of This Article

Publication Name

World Journal of Gastroenterology

ISSN

1007-9327

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Topic Highlight

World J Gastroenterol. Jun 21, 2014; 20(23): 7089-7103
Published online Jun 21, 2014. doi: 10.3748/wjg.v20.i23.7089

Open in New Tab Full Size Figure Download Figure

Figure 1 The hepatitis C virus-associated dysmetabolic syndrome (modified from ref[10]). HCV: Hepatitis C virus.

Open in New Tab Full Size Figure Download Figure

Figure 2 General steatogenic pathways associated with hepatitis C virus infection (reprinted from ref[9]). HCV: Hepatitis C virus.

Open in New Tab Full Size Figure Download Figure

Figure 3 Genotype-specific steatogenic pathways associated with hepatitis C virus infection[63-69]. The negative sign (-) indicates inhibited and the positive sign (+) indicates stimulated pathway. FAO: Fatty acid oxidase; FAS: Fatty acid synthase; FFA: Free fatty acids; MTP: Microsomal triglyceride transfer protein; PPAR-alpha: Peroxisome proliferator-activated receptor alpha; PTEN: Phosphatase and tensin homologue; ROS: Reactive oxygen species; SOCS3: Suppressor of cytokine signaling; SREBPs: Sterol regulatory element-binding proteins; TNF alpha: Tumor necrosis factor alpha; HCV: Hepatitis C virus.

Citation: Lonardo A, Adinolfi LE, Restivo L, Ballestri S, Romagnoli D, Baldelli E, Nascimbeni F, Loria P. Pathogenesis and significance of hepatitis C virus steatosis: An update on survival strategy of a successful pathogen. World J Gastroenterol 2014; 20(23): 7089-7103
URL: https://www.wjgnet.com/1007-9327/full/v20/i23/7089.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i23.7089